Genetic mutation may disable fat sensors, raise obesity risk

02/19/2012 | eMaxHealth.com

Mice carrying a genetic defect making them deficient in the fat-sensing GPR-120 protein gained more weight and developed higher fasting plasma glucose and insulin levels than their normal wild type counterparts when fed a high-fat diet, a study in Nature found. An analysis in humans showed one particular mutation in the gene sequence coding for GPR-120 disables its fat-sensing capabilities, resulting in a 60% increase in obesity risk, researchers said.

View Full Article in:

eMaxHealth.com

Published in Briefs:

SmartBrief Job Listings for Health Care

Job Title Company Location
Consumer Directed Health Plan (CDH) Product Offering Manager
Blue Cross Blue Shield MA
Quincy, MA
Actuary
Meridian Health Plan
Detroit, MI
ISHN - Chief Compliance Officer
Mountain States Health Alliance
Johnson City, TN
President/Chief Executive Officer
MedCost
Winston-Salem, NC
Director, Payer Marketing
Avalere Health
Washington, DC