Scientists find potential drug target to stop HIV infection

10/2/2013 | Medical News Today

Cambridge University researchers suggest that a mutation of the adhesion and degranulation-promoting adaptor protein could help create ways to fight off HIV without weakening the immune system. The study, which appears in the journal Retrovirology, showed that the mutant form of ADAP regulates infection and interferes with HIV-1 spread within T cells and between infected and uninfected T cells. Researchers say that giving HIV patients T cells with mutant ADAP would effectively block these processes.

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