U.S. and Japanese researchers found that increasing expression in the proteins TR2 and TR4 boosted the level of fetal hemoglobin and reduced organ damage in mice with sickle cell disease. Some patients born with higher levels of fetal hemoglobin show less severe complications and higher survival rates, leading researchers to conclude that increasing fetal hemoglobin is one way to treat the disease, according to the study published in the Proceedings of the National Academy of Sciences.

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