Researchers found that astrocytes from individuals with Parkinson's disease had significantly elevated alpha-synuclein levels, as well as increased inflammatory reactivation sensitivity, compared with control astrocytes. The findings in Scientific Reports also showed that astrocytes with Parkinson's mutations had reduced mitochondrial DNA copies and functioning, as well as reduced lysophosphatidylethanolamine levels associated with Parkinson's, suggesting that astrocytes play a role in Parkinson's progression, researchers said.
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